Alpurinol Tablet

Description

what is alpurinol tablet powder？

Allopurinol powder, White or off white crystalline powder, very slightly soluble in water or ethanol. Insoluble in chloroform or ether; Soluble in sodium hydroxide or potassium hydroxide. This product and its metabolites can inhibit xanthine oxidase, so that hypoxanthine and xanthine cannot be converted into uric acid, that is, uric acid synthesis is reduced, thereby reducing the concentration of uric acid in blood, reducing the deposition of urate in bones, joints and kidneys, and a drug that can inhibit uric acid synthesis. This product can inhibit the activity of liver drug enzymes. This product is only for scientific research.

Allopurinol is a drug that inhibits the synthesis of uric acid. Allopurinol and its metabolites can prevent hypoxanthine and xanthine from being metabolized into uric acid, thus reducing the generation of uric acid, reducing the content of uric acid in blood and urine below the solubility level, preventing uric acid from forming crystals, depositing in joints and other tissues, causing the onset of gout, and also helping to re dissolve uric acid crystals in gout patients' tissues.

Clinical use:

1. Primary and secondary hyperuricemia, especially hyperuricemia with excessive uric acid production, is also used for hyperuricemia with renal insufficiency;

2. For the treatment of gout, suitable for recurrent or chronic gout. For patients with gouty nephropathy, it can alleviate symptoms and reduce the formation of uric acid stones in the kidney;

3. Gout stone;

4. For uric acid kidney stones and / or uric acid nephropathy.

Allopurinol has the effect of reducing uric acid mainly by inhibiting the synthesis of uric acid. Allopurinol and its metabolites can inhibit xanthine oxidase, so as to prevent hypoxanthine and xanthine from being metabolized into uric acid, reduce the production of uric acid, reduce the content of uric acid in blood below the solubility, prevent uric acid from forming crystals and depositing in joints and other tissues, and contribute to the re dissolution of uric acid crystals in gout patients. Therefore, it can be mainly used in patients with hyperuricemia. The blood uric acid concentration begins to decline 24 hours after taking this drug, and the decline is most obvious in 2-4 weeks. Allopurinol can be mainly used in clinical practice for primary or secondary hyperuricemia, especially hyperuricemia caused by excessive uric acid production. It can also be used for recurrent attacks or chronic gout. It can also be used in patients with gout stone. It has a certain effect on uric acid kidney stones and uric acid nephropathy. It can also be used in hyperuricemia in patients with renal insufficiency.

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